Buscar en
Revista Colombiana de Cardiología
Toda la web
Inicio Revista Colombiana de Cardiología Falla cardiaca e hiperaldosteronismo primario Presentación de un caso
Journal Information
Vol. 19. Issue 3.
Pages 142-147 (May - June 2012)
Share
Share
Download PDF
More article options
Vol. 19. Issue 3.
Pages 142-147 (May - June 2012)
Open Access
Falla cardiaca e hiperaldosteronismo primario Presentación de un caso
Heart failure and primary hyperaldosteronism Case report
Visits
2958
Juan M. Camargo1,
Corresponding author
camargojuan2007@gmail.com

Correspondencia: Calle 97 No. 70-89 Interior 4, Apto 904, Tel (571) 645 8434, Bogotá, DC., Colombia.
, Fernán del C. Mendoza1, Efraín A. Gómez1, Rubén D. Luna1, Claudia Méndez1
1 Fundación Clínica Abood Shaio. Bogotá, DC., Colombia
This item has received

Under a Creative Commons license
Article information

La insuficiencia cardiaca es un síndrome asociado con alta morbilidad y mortalidad, principalmente debido a episodios de agudización o descompensación. La cardiopatía hipertensiva es una etiología de la insuficiencia cardiaca con alta prevalencia en el mundo. El hiperaldosteronismo primario es una causa de hipertensión con incidencia creciente, que, independiente de la hipertensión, puede desencadenar miocardiopatía con todas sus consecuencias.

En este artículo se presenta el caso de un paciente de cincuenta años con insuficiencia cardiaca agudizada con disfunción sistólica, asociada a hipertensión resistente y como patología de base un estado con hipersecreción de aldosterona (hiperaldosteronismo primario).

Palabras clave:
insuficiencia cardiaca aguda
hipertensión resistente
miocardiopatía

Heart failure is a syndrome associated with high morbidity and mortality, mainly due to episodes of exacerbation or decompensation. Hypertensive heart disease is a cause of heart failure with a high prevalence in the world. Primary hyperaldosteronism is a cause of hypertension with increasing incidence, which, independent of hypertension, can lead to cardiomyopathy with all its consequences.

This article presents the case of a fifty years old male patient with acute heart failure exacerbated with systolic dysfunction, associated with resistant hypertension and having as underlying pathology a condition of aldosterone hypersecretion (primary hyperaldosteronism).

Key words:
acute heart failure
resistant hypertension
cardiomyopathy
Full text is only aviable in PDF
Bibliografía
[1.]
K. Dickstein, A. Cohen-Solal, G. Filippatos, et al.
The task force for the diagnosis and treatment of acute and chronic heart failure 2008 of the European Society of Cardiology.
Eur Heart J, 29 (2008), pp. 2388-2442
[2.]
W.F. Young.
Primary aldosteronism: renaissance of a syndrome.
Clin Endocrinol (Oxf), 66 (2007), pp. 607-618
[3.]
P.M. Stewart.
Mineralocorticoid hypertension.
Lancet, 353 (1999), pp. 1341-1347
[4.]
L. Mosso, C. Carvajal, A. González, et al.
Primary aldosteronism and hypertensive disease.
Hypertension, 42 (2003), pp. 161-165
[5.]
C.E. Fardella, L. Mosso, C. Gómez-Sánchez, et al.
Primary hyperaldosteronism in essential hypertensives: prevalence, biochemical profile and molecular biology.
J Clin Endocrinol Metab, 85 (2000), pp. 1863-1867
[6.]
S. Douma, K. Petidis, M. Doumas, et al.
Prevalence of primary hyperaldosteronism in resistant hypertension: a retrospective observational study.
Lancet, 371 (2008), pp. 1921-1926
[7.]
R. Rocha, J.W. Funder.
The pathophysiology of aldosterone in the cardiovascular system.
Ann N Y Acad Sci, 970 (2002), pp. 89-100
[8.]
P. Milliez, X. Girerd, P.F. Plouin, et al.
Evidence for an increased rate of cardiovascular events in patients with primary aldosteronism.
J Am Coll Cardiol, 45 (2005), pp. 1243-1245
[9.]
K.T. Weber.
Aldosterone in congestive heart failure.
N Engl J Med, 345 (2001), pp. 1689-1697
[10.]
N. Laleve‘e, M. Rebsamen, S. Barrere-Lemaire, et al.
Aldosterone increases Ttype calcium channel expression and in vitro beating frequency in neonatal rat cardiomyocytes.
Cardiovasc Res, 67 (2005), pp. 216-224
[11.]
M.L. Muiesan, M. Salvetti, A. Paini, et al.
Inappropriate left ventricular mass in patients with primary aldosteronism.
Hypertension, 52 (2008), pp. 529-534
[12.]
P. Mulatero, M. Stowasser, K.C. Loh, et al.
Increased diagnosis of primary aldosteronism, including surgically correctable forms, in centers from five continents.
J Clin Endocrinol Metab, 89 (2004), pp. 1045-1050
[13.]
C. Mattsson, W.F. Young Jr..
Primary aldosteronism: diagnostic and treatment strategies.
Nat Clin Pract Nephrol, 2 (2006), pp. 198-208
[14.]
M.K. Walz, R. Gwosdz, S.L. Levin, et al.
Retroperitoneoscopic adrenalectomy in Conn‘s syndrome caused by adrenal adenomas or nodular hyperplasia.
World J Surg, 32 (2008), pp. 847-853
[15.]
J.W. Funder, R.M. Carey, C. Fardella, et al.
Case detection, diagnosis and treatment of patients with primary aldosteronism: An Endocrine Society Clinical Practice Guideline.
J Clin Endocrinol Metab, 93 (2008), pp. 3266-3281
[16.]
P. Mulatero, S. Monticone, C. Bertello, et al.
Evaluation of primary aldosteronism.
Curr Opin Endocrinol Diabetes Obes, 17 (2010), pp. 188-193
[17.]
M. Boscaro, V. Ronconi, F. Turchi, et al.
Diagnosis and management of primary aldosteronism.
Curr Opin Endocrinol Diabetes Obes, 15 (2008), pp. 332-338
[18.]
G. Giacchetti, P. Mulatero, F. Mantero, et al.
Primary aldosteronism, a major form of low renin hypertension: from screening to diagnosis.
Trends Endocrinol Metab, 19 (2008), pp. 104-108
[19.]
W.F. Young, A.W. Stanson, G.B. Thompson, et al.
Role for adrenal venous sampling in primary aldosteronism.
Surgery, 136 (2004), pp. 1227-1235
[20.]
P.E. Gleason, M.H. Weinberger, J.H. Pratt, et al.
Evaluation of diagnostic tests in the differential diagnosis of primary aldosteronism: unilateral adenoma versus bilateral micronodular hyperplasia.
J Urol, 150 (1993), pp. 1365-1368
[21.]
C. Tresallet, H. Salepcioglu, G. Godiris-Petit, et al.
Clinical outcome after laparoscopic adrenalectomy for primary hyperaldosteronism: The role of pathology.
Surgery, 148 (2010), pp. 129-134
[22.]
C.G. Brilla, R. Pick, L.B. Tan, et al.
Remodeling of the rat right and left ventricle in experimental hypertension.
Circ Res, 67 (1990), pp. 1355-1364
[23.]
B. Schwartzkopff, B.E. Strauer.
Squeezing tubes: a case of remodeling and regulation: coronary reserve in hypertensive heart disease.
Cardiovasc Res, 40 (1998), pp. 4-8
[24.]
R. Rocha, C.T. Stier.
Pathophysiological effects of aldosterone in cardiovascular tissues.
Trends Endocrinol Metab, 12 (2001), pp. 308-314
[25.]
American Association of clinical Endocrinologists medical guidelines for clinical practice for the diagnosis, treatment of hypertension.
AACE Hypertension task force.
Endocr Pract, 12 (2006), pp. 193-222
[26.]
R.J. Weigel, S.A. Wells, J.C. Gunnells, et al.
Surgical treatment of primary hyperaldosteronism.
Ann Surg, 219 (1994), pp. 347-352
[27.]
A.M. Sawka, W.F. Young, G.B. Thompson, et al.
Primary aldosteronism: factors associated with normalization of blood pressure after surgery.
Ann Intern Med, 135 (2001), pp. 258-261
[28.]
B. Pitt, F. Zannad, W.J. Remme, et al.
The effect of spironolactone on morbidity and mortality in patients with severe heart failure.
N Engl J Med, 341 (1999), pp. 709-717
[29.]
F. Zannad, F. Alla, B. Dousset, et al.
Limitation of excessive extracellular matrix turnover may contribute to survival benefit of spironolactone therapy in patients with congestive heart failure: insights from the randomized aldactone evaluation study.
Circulation, 102 (2000), pp. 2700-2706
[30.]
A. Karagiannis, K. Tziomalos, A. Papageorgiou, et al.
Spironolactone versus eplerenone for the treatment of idiopathic hyperaldosteronism.
Expert Opin Pharmaco Ther, 9 (2008), pp. 509-515
Copyright © 2012. Sociedad Colombiana de Cardiología y Cirugía Cardiovascular
Article options
Tools